Dr. Sekar joined The University of Findlay, School of Pharmacy in July 2007 as Associate Professor in Pharmaceutical Sciences. Dr. Sekar has several years of pharmacy practice experience at the University of Cincinnati hospital, where he provided pharmacy services in both central and decentralized areas of practice. He brings with him over 20 years of research experience in molecular pharmacology. His area of specialization is in cell communication and has made several important contributions in the area of phosphoinositide signaling.

At The University of Findlay, he hopes to incorporate his extensive research and pharmacy experience in his pharmacology teaching curriculum and provide electives in research electives that should help pharmacists interested in pursuing research careers

MEMBERSHIPS

American Association for Advancement of Science
American Pharmaceutical Association
American Association for Consultant Pharmacists
Ohio Pharmacist Association

LICENSED PHARMACIST

Ohio
Florida
Washington

RESEARCH GRANT SUPPORT

National Institute of Health: 1992-1999
American Heart Association: 1991-1993
American Diabetes Association: 1989-1991
Cystic Fibrosis Foundation: 1990-1991

Sekar MC, Scott ED, Sambandam V, Berry RE. Demonstration of the presence of cyclic inositol phosphohydrolase in human urine. Biochem Mol Med 1997:62:95-100.

Sekar MC, Sambandam V, Grizzle WE, and McDonald JM. Dissociation of cyclic inositol phosphate hydrolase activity from annexin III. J Biol Chem 1996;271:8295-8299.

Chen P, Xie H, Sekar MC, Gupta K and Wells A. Epidermal growth factor receptor-mediated cell mobility: Phospholipase C activity is required, but MAP kinase activity is not sufficient for induced cell mobility. J Cell Biol 1994;127:847-857.

Lanas A, Sekar MC and Hirschowitz BI. Objective evidence of Aspirin use in both ulcer and non-ulcer upper and lower gastrointestinal bleeding. Gastroenterology 1992;103:862-869.

Sekar MC, Uemura N, Coy DH, Hirschowitz BI and Dickinson KEJ. Bombesin, neuromedin B and neuromedin C interact with a common rat pancreatic phosphoinositide-coupled receptor but are differentially regulated by guanine nucleotides. Biochem J 1991;280:163-169.

Sekar MC, Yang M, Meezan E and Pillion DJ. Angiotensin II and bradykinin stimulate phosphoinositide breakdown in intact rat kidney glomeruli but not in proximal tubules. Glomerular response modulated by phorbol ester. Biochem Biophys Res Commun 1990;166:373-379.

Sekar MC, Dixon JF and Hokin LE. The formation of Inositol 1,2-Cyclic 4,5-trisphosphate and inositol 1,2-Cyclic 4-Bisphosphate on stimulation of mouse pancreatic minilobules with carbamylcholine. J Biol Chem 1987;262:340-344.

Rana RS, Sekar MC, Mertz RJ, Hokin LE and MacDonald MJ. Potentiation by glucose metabolites of inositol trisphosphate-induced calcium mobilization in permeabolized rat pancreatic islets. J Biol Chem 1987;262:13567-13570.

Sekar MC and Roufogalis BD. Muscarinic receptor stimulation of polyphosphoinositide breakdown in guinea pig smooth muscle: Amplification by lithium. Biochem J 1984;223:527-531.